Posts Tagged ‘professional’

Share Your Research on a Blog

How can researchers provide information about their studies in ways that would be useful and interesting to prospective and current research participants? With that question in my mind, MethodSpace’s Janet Salmons began to explore the potential for blogs to recruit and inform participants. As with almost any online exploration, she discovered a much broader potential for blogs in the academic world.

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Brexit McCarthyism, Universities PLC and the Erosion of Academic Freedom in the UK

Last week the UK academic world was abuzz about Chris Heaton- Harris’ letter on Brexit. This week it’s not. This lack of lasting public interest in threats to academic freedom is lamentable even as the corporatization of the nation’s universities is the biggest threat facing academic life and flies completely under the public’s radar, argues Daniel Nehring.

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Contexts, Practices and Challenges: Critical Insights from Continuing Professional Education: New Directions for Adult and Continuing Education, Number 151

  Lifelong learning has become essential not only for professionals, but also for those they serve. Continuing professional education (CPE), an umbrella term used to describe the continuum of formal, nonformal, and informal learning opport…

Macrophages redirect phagocytosis by non-professional phagocytes and influence inflammation

Professional phagocytes (such as macrophages) and non-professional phagocytes (such as epithelial cells) clear billions of apoptotic cells and particles on a daily basis. Although professional and non-professional macrophages reside in proximity in most tissues, whether they communicate with each other during cell clearance, and how this might affect inflammation, is not known. Here we show that macrophages, through the release of a soluble growth factor and microvesicles, alter the type of particles engulfed by non-professional phagocytes and influence their inflammatory response. During phagocytosis of apoptotic cells or in response to inflammation-associated cytokines, macrophages released insulin-like growth factor 1 (IGF-1). The binding of IGF-1 to its receptor on non-professional phagocytes redirected their phagocytosis, such that uptake of larger apoptotic cells was reduced whereas engulfment of microvesicles was increased. IGF-1 did not alter engulfment by macrophages. Macrophages also released microvesicles, whose uptake by epithelial cells was enhanced by IGF-1 and led to decreased inflammatory responses by epithelial cells. Consistent with these observations, deletion of IGF-1 receptor in airway epithelial cells led to exacerbated lung inflammation after allergen exposure. These genetic and functional studies reveal that IGF-1- and microvesicle-dependent communication between macrophages and epithelial cells can critically influence the magnitude of tissue inflammation in vivo.

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